p53 is a tumor suppressor gene. How does this gene normally prevent cancer in your cells?(1 point)

Question

Responses

A transcription factor binds to the promoter of genes that stop cells from from duplicating themselves when DNA is damaged
A transcription factor binds to the promoter of genes that stop cells from from duplicating themselves when DNA is damaged

It produces a repressor protein that binds to an operon and stops further gene transcription.
It produces a repressor protein that binds to an operon and stops further gene transcription.

A transcription factor binds RNA polymerase, stopping the cell from getting large enough to be able to split in half and continue making cancerous cells.
A transcription factor binds RNA polymerase, stopping the cell from getting large enough to be able to split in half and continue making cancerous cells.

It stops RNA polymerase from binding to any DNA, halting all replication in the cell.

Answer 1

The correct response is:

A transcription factor binds to the promoter of genes that stop cells from duplicating themselves when DNA is damaged.

p53 functions primarily as a transcription factor that regulates the expression of genes involved in the cell cycle, DNA repair, and apoptosis (programmed cell death). When DNA is damaged, p53 activates the expression of genes that can halt the cell cycle, thereby allowing time for the damage to be repaired or triggering apoptosis if the damage is irreparable. This protective mechanism is crucial in preventing the development of cancer.