Question
Rotenone is a toxic substance derived from several tropical and subtropical plants. It is used as a pesticide to control certain populations of insects and fish. Rotenone interferes with the electron transport chain in the mitochondria of the cells of those organisms.
If the electron transport chain does not function properly, how does that impact the cells?
Responses
It prevents the release of carbon dioxide by a cell.
It prevents the release of carbon dioxide by a cell.
It causes the accumulation of high levels of lactic acid.
It causes the accumulation of high levels of lactic acid.
It prevents the production of ATP during aerobic respiration.
It prevents the production of ATP during aerobic respiration.
It causes the cell to dehydrate by losing large amounts of water.
If the electron transport chain does not function properly, how does that impact the cells?
Responses
It prevents the release of carbon dioxide by a cell.
It prevents the release of carbon dioxide by a cell.
It causes the accumulation of high levels of lactic acid.
It causes the accumulation of high levels of lactic acid.
It prevents the production of ATP during aerobic respiration.
It prevents the production of ATP during aerobic respiration.
It causes the cell to dehydrate by losing large amounts of water.
Answers
GPT-4o mini
The most accurate impact of rotenone's interference with the electron transport chain is:
**It prevents the production of ATP during aerobic respiration.**
Rotenone blocks the flow of electrons in the electron transport chain, which is essential for the process of oxidative phosphorylation, a key component of aerobic respiration. This disruption inhibits the generation of ATP, the primary energy currency of the cell, leading to energy depletion and potentially cell death. While there may be some impacts on lactic acid levels due to the switch to anaerobic metabolism in the absence of adequate ATP, the direct and primary effect of rotenone is its interference with ATP production.
**It prevents the production of ATP during aerobic respiration.**
Rotenone blocks the flow of electrons in the electron transport chain, which is essential for the process of oxidative phosphorylation, a key component of aerobic respiration. This disruption inhibits the generation of ATP, the primary energy currency of the cell, leading to energy depletion and potentially cell death. While there may be some impacts on lactic acid levels due to the switch to anaerobic metabolism in the absence of adequate ATP, the direct and primary effect of rotenone is its interference with ATP production.